Deck 26: Diabetes Mellitus

ملء الشاشة (f)
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سؤال
Diabetic ketoacidosis is characterized by severe ketosis, acidosis, and ____.

A) renal failure
B) hallucinations
C) hyperglycemia
D) myocardial ischemia
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سؤال
What best describes polydipsia?

A) excessive thirst
B) excessive urination
C) increased glucose in the urine
D) excessive hunger
سؤال
Normal fasting plasma glucose levels are approximately ____ mg/dL.

A) 60 to 74
B) 75 to 100
C) 110 to 125
D) 126 to 140
سؤال
Which of the following is NOT a feature of type 2 diabetes?

A) insulin resistance
B) autoimmune disease
C) obesity is a causative factor
D) some cases require insulin therapy
سؤال
Which condition is a microvascular complication of diabetes?

A) peripheral vascular disease
B) diabetic retinopathy
C) intermittent claudication
D) hypertriglyceridemia
سؤال
People with type 1 diabetes need exogenous insulin because they ____.

A) have become insulin-resistant
B) have developed hyperinsulinemia
C) no longer synthesize insulin
D) digest insulin with gastrointestinal enzymes
سؤال
The renal threshold is described as ____.

A) a decrease in erythropoietin production because of insulin imbalance
B) nutrient depletion in the renal tubules caused from deficient insulin
C) hemoglobin's exposure to glucose within the kidneys
D) the concentration at which the kidneys begin to pass glucose into the urine
سؤال
The oral glucose tolerance test uses a glucose load of ____ grams.

A) 25
B) 50
C) 75
D) 100
سؤال
Glycosuria usually occurs when plasma glucose concentration exceeds ____ mg/dL.

A) 140
B) 160
C) 180
D) 200
سؤال
A fasting blood glucose level above ____ mg/dL is classified as diabetes.

A) 90
B) 100
C) 116
D) 126
سؤال
Which ethnic population has the lowest relative risk for developing type 2 diabetes?

A) Pacific Islanders
B) Asian Indians
C) European Americans
D) Native Americans
سؤال
Diabetic ketoacidosis can develop when blood glucose is greater than ____ mg/dL.

A) 250
B) 450
C) 650
D) 700
سؤال
About ____ percent of persons with diabetes are unaware that they have it.

A) 7
B) 17
C) 24
D) 37
سؤال
The pancreatic hormone that promotes gluconeogenesis in the liver is ____.

A) glycogen
B) estrogen
C) glucagon
D) leptin
سؤال
Type 1 diabetes accounts for ____ percent of diabetes cases in the United States.

A) 5 to 10
B) 15 to 25
C) 50 to 75
D) 90 to 95
سؤال
Using the "intensive therapy" approach, blood glucose is monitored at least ____ time(s) a day for people with type 1 diabetes.

A) one
B) two
C) three
D) four
سؤال
What is the standard treatment for hyperosmolar hyperglycemic syndrome?

A) intravenous fluid and electrolyte replacement and insulin therapy
B) intravenous administration of ketones
C) surgical resection of the pancreas
D) a low-carbohydrate, low-fat diet
سؤال
Which is NOT a sign or symptom of diabetic neuropathy?

A) Numbness or tingling in extremities
B) gastroparesis
C) bladder dysfunction
D) diarrhea
سؤال
A blood glucose level between 100 and 125 mg/dL after an 8-hour fast would be classified as ____.

A) impaired fasting glucose
B) hypoglycemia
C) impaired glucose tolerance
D) normal fasting glucose
سؤال
Symptoms of hypoglycemia include ____.

A) sweating and heart palpitations
B) increased thirst and polyuria
C) acetone breath
D) warm, flushed skin
سؤال
Mrs. Barclay has type 2 diabetes and you are preparing her for discharge. She tells you that she loves red beans and rice and knows that she must eliminate them from her diet because they will elevate her blood glucose level. You should explain to her that ____.

A) on a carbohydrate-counting meal plan, these foods in recommended portion sizes can fit within her carbohydrate allowance at meals
B) red beans are eliminated because they are high in complex carbohydrates
C) red beans are high in water-soluble fiber and should be avoided
D) peas are a better choice than red beans
سؤال
As per the carbohydrate-counting meal plan, how many grams of carbohydrates is in a snack that consists of 2 cups of ice cream and a small apple?

A) 60 grams
B) 75 grams
C) 45 grams
D) 15 grams
سؤال
The newborn of a mother with diabetes is at greater risk of which metabolic condition?

A) hypocalcemia
B) sepsis
C) transient tachypnea
D) congenital heart disease
سؤال
Short-acting insulin begins to act ____ after it is injected.

A) 15 to 20 minutes
B) 30 to 60 minutes
C) 1 to 3 hours
D) 2 to 4 hours
سؤال
Approximately ____ percent of nondiabetic women in the United States develop gestational diabetes.

A) 4 to 14
B) 14 to 24
C) 24 to 34
D) 34 to 44
سؤال
Hemoglobin A 1c can best be described as a(n) ____.

A) by-product of fat metabolism
B) reflection of glycemic control over the preceding 2 to 3 months
C) end-product of protein metabolism formed in the liver
D) summary of hemoglobin rates for type 1 diabetes
سؤال
Women with gestational diabetes are at greater risk of which condition later in life?

A) obesity
B) type 2 diabetes
C) metabolic syndrome
D) chronic kidney disease
سؤال
Insulin is most often administered by ____.

A) subcutaneous injection
B) oral administration
C) intramuscular injection
D) intravenous administration
سؤال
Which antidiabetic drug is classified as a biguanide?

A) acarbose
B) pramlintide
C) metformin
D) nateglinide
سؤال
The goal of diabetes treatment is an HbA 1c value under ____ percent.

A) 5
B) 7
C) 9
D) 10
سؤال
What is an advantage seen with intensive therapy for patients with type 1 diabetes?

A) delayed progression of retinopathy, nephropathy, and neuropathy
B) less weight gain
C) greater stability noted in fructosamine results
D) fewer incidents of severe hypoglycemia
سؤال
Which recommendation is not advised for pregnant women with diabetes?

A) insulin therapy
B) 10% weight loss
C) physical activity
D) low-carbohydrate breakfast
سؤال
What maternal complication has been associated with uncontrolled diabetes during pregnancy?

A) preeclampsia
B) obesity
C) hyperemesis gravidarum
D) miscarriage
سؤال
Hyperglycemia that results from the release of counterregulatory hormones following nighttime hypoglycemia is known as ____.

A) fasting hyperglycemia
B) rebound hyperglycemia
C) dawn phenomenon
D) nocturnal hyperglycemia
سؤال
What test may be used to determine glycemic control over the preceding 2- to 3-week period?

A) fructosamine
B) ketone
C) glucose tolerance
D) insulin antibody
سؤال
Mr. Lilly, a 42-year-old male, tells you that he used to take "pills" for his diabetes but is now taking insulin. What is the most likely reason that he no longer takes oral antidiabetic agents?

A) Oral agents are effective only in type 1 diabetes.
B) His body now stimulates enough insulin to meet his needs.
C) Oral agents suppress insulin release.
D) The oral agents were not effectively controlling his blood glucose levels.
سؤال
Mr. Jacobs has high blood lipids. The physician would instruct him to limit dietary intake of which substance as an added sweetener but not from whole foods (i.e., fruits and vegetables)?

A) fructose
B) sucrose
C) glucose
D) sucralose
سؤال
Which intervention is most appropriate as part of sick-day management of diabetes?

A) Discontinue all antidiabetic medications and insulin.
B) Measure blood glucose and urine ketones once a day.
C) Discontinue antidiabetic drugs and only use insulin.
D) Consume the usual diet if possible.
سؤال
As per the carbohydrate-counting meal plan, how many grams of carbohydrates is in 2 cups of brown rice and a cup of red beans?

A) 100 grams
B) 120 grams
C) 150 grams
D) 175 grams
سؤال
What is a metabolic effect that may occur with taking thiazolidinediones for glycemic control?

A) fluid retention
B) weight loss
C) allergic skin reactions
D) vitamin B 12 deficiency
سؤال
Which patient would have the most risk factors for gestational diabetes?

A) Rita, who has a prepregnancy body mass index (BMI) of 24
B) Zhu, who just moved to the United States from China
C) Amy, who once had an 8-pound baby
D) Sarah, whose father has diabetes
سؤال
An estimated ____ percent of adults in the United States meet the criteria for metabolic syndrome.

A) 6
B) 12
C) 23
D) 34
سؤال
Which antidiabetic drug is sometimes prescribed for type 2 diabetic women during pregnancy?

A) glimepiride
B) nateglinide
C) repaglinide
D) glyburide
سؤال
56-60. Short Case-Study Questions  
Michael Fern is a 21-year-old college student majoring in electrical engineering. His medical history is unremarkable. He comes to his family doctor for the second time this month because of symptoms of lethargy, fatigue, weight loss, and frequent thirst and urination.
After 6 months, Michael's endocrinologist orders an insulin pump. Which best describes the use of an insulin pump with type 1 diabetes?

A) Hyperglycemia is not a concern with the insulin pump.
B) Hypoglycemia can occur with insulin treatment.
C) Michael does not need to follow a modified diet.
D) Michael no longer needs to worry about exercising.
سؤال
Diagnostic criteria for metabolic syndrome include a fasting plasma glucose of ____ mg/dL or higher.

A) 80
B) 90
C) 100
D) 110
سؤال
Diagnostic criteria for metabolic syndrome include blood pressure of ____ mm Hg or higher.

A) 110/80
B) 130/85
C) 150/90
D) 170/95
سؤال
56-60. Short Case-Study Questions  
Michael Fern is a 21-year-old college student majoring in electrical engineering. His medical history is unremarkable. He comes to his family doctor for the second time this month because of symptoms of lethargy, fatigue, weight loss, and frequent thirst and urination.
The registered dietitian visits with Michael to instruct him on his diabetic diet. What is important for Michael to know about consuming alcohol?

A) He should not consume any alcohol.
B) He should consume food when he ingests alcoholic beverages to avoid hypoglycemia.
C) He should limit his food intake prior to consuming alcohol in order to avoid hyperglycemia.
D) Excessive alcohol intake will likely cause severe hypoglycemia.
سؤال
56-60. Short Case-Study Questions  
Michael Fern is a 21-year-old college student majoring in electrical engineering. His medical history is unremarkable. He comes to his family doctor for the second time this month because of symptoms of lethargy, fatigue, weight loss, and frequent thirst and urination.
Michael is rushed to the hospital after a simple blood test determines a blood glucose of 600 mg/dL. He is most likely suffering from ____.

A) hypoglycemia
B) hyperglycemia
C) dehydration
D) infection
سؤال
Diagnostic criteria for metabolic syndrome include waist circumference of greater than ____ inches in women.

A) 33
B) 35
C) 38
D) 40
سؤال
Inflammation of endothelial tissue, obesity, and insulin resistance can all contribute to blood clots by promoting the increased production of procoagulant proteins such as ____.

A) hirudin
B) fibrinogen
C) dicoumarol
D) andexanet alfa
سؤال
Macrosomia develops as a result of ____.

A) insulin's stimulatory effect on fat synthesis
B) excessive insulin production by the fetal pancreas
C) poor maternal kidney function due to excess glucose
D) the enzymatic glycation of serum proteins
سؤال
56-60. Short Case-Study Questions  
Michael Fern is a 21-year-old college student majoring in electrical engineering. His medical history is unremarkable. He comes to his family doctor for the second time this month because of symptoms of lethargy, fatigue, weight loss, and frequent thirst and urination.
Michael's ketoacidosis leads to a diagnosis of type 1 diabetes. Which medication will Michael need to take to treat his diabetes?

A) sulfonylureas
B) metformin
C) insulin injections
D) alpha-glucosidase inhibitors
سؤال
56-60. Short Case-Study Questions  
Michael Fern is a 21-year-old college student majoring in electrical engineering. His medical history is unremarkable. He comes to his family doctor for the second time this month because of symptoms of lethargy, fatigue, weight loss, and frequent thirst and urination.
Michael has been diagnosed with diabetic ketoacidosis. In addition to severely elevated blood glucose levels, what other symptom is associated with this condition?

A) fruity odor on the breath
B) fluid retention
C) high blood pressure
D) vision loss
سؤال
Regina is at her first prenatal visit after recently discovering that she is pregnant. She is a low-risk patient with no history of diabetes. At what point will her physician test Regina for gestational diabetes?

A) 12 to 15 weeks
B) 16 to 20 weeks
C) 24 to 28 weeks
D) 29 to 32 weeks
سؤال
Diagnostic criteria for metabolic syndrome include high-density lipoprotein (HDL) levels of less than ____ mg/dL in men.

A) 30
B) 35
C) 40
D) 50
سؤال
Diagnostic criteria for metabolic syndrome include very-low-density lipoprotein (VLDL) levels of ____ mg/dL or higher.

A) 100
B) 125
C) 150
D) 175
سؤال
Emily has gestational diabetes and a BMI of 33. What nutrition recommendation should be made for Emily for weight control during her pregnancy?

A) Strive to lose 11 to 14 lb by the end of her pregnancy.
B) Increase carbohydrate intake to 55 to 60% of total energy.
C) Reduce kcalorie intake by 30% to slow further weight gain.
D) Continue with a normal diet and strive to lose weight after pregnancy.
سؤال
How does obesity lead to the development of hypertriglyceridemia?

A) Insulin causes adipose cells to store more triglycerides.
B) HDL competes with VLDL for cholesterol binding sites.
C) The liver produces more VLDL in response to fatty acids released by insulin-resistant adipose cells.
D) Fatty acids in the bloodstream resist excretion by HDL.
سؤال
The hyperinsulinemia that typically accompanies obesity promotes sodium reabsorption in the kidneys, resulting in fluid retention and ____.

A) increased abdominal obesity
B) hypertension
C) atherosclerosis
D) hypertriglyceridemia
سؤال
What dietary recommendation is advised for individuals with hypertriglyceridemia?

A) Reduce intake of added sugars and refined grain products.
B) Decrease sodium intake and improve intake of dairy products.
C) E liminate polyunsaturated fatty acids in the diet.
D) Reduce kcalorie intake by 45% daily.
سؤال
61-65. Short Case Study Questions  
Ruth Ann Glebowski is a 40-year-old woman. She has a medical history of gestational diabetes, high cholesterol, hypothyroidism, and depression. She is 5 ft 4 in. tall and weighs 190 lb. Recently she has made an appointment with her physician since she has been having excessive hunger and frequent urination.
Ruth Ann's doctor can best evaluate long-term diabetic compliance by checking her ____.

A) fasting blood glucose
B) urine ketones
C) blood pressure
D) glycated hemoglobin
سؤال
Match between columns
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
polydipsia
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
type 1 diabetes
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
prediabetes
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
adiponectin
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
glycated hemoglobin
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
type 2 diabetes
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
metabolic syndrome
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
macrosomia
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
polyuria
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
hyperosmolar hyperglycemic syndrome
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
polyphagia
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
diabetic nephropathy
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
diabetic retinopathy
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
diabetic neuropathy
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
advanced glycation end products
excessive urine production
polydipsia
excessive urine production
type 1 diabetes
excessive urine production
prediabetes
excessive urine production
adiponectin
excessive urine production
glycated hemoglobin
excessive urine production
type 2 diabetes
excessive urine production
metabolic syndrome
excessive urine production
macrosomia
excessive urine production
polyuria
excessive urine production
hyperosmolar hyperglycemic syndrome
excessive urine production
polyphagia
excessive urine production
diabetic nephropathy
excessive urine production
diabetic retinopathy
excessive urine production
diabetic neuropathy
excessive urine production
advanced glycation end products
excessive appetite or eating
polydipsia
excessive appetite or eating
type 1 diabetes
excessive appetite or eating
prediabetes
excessive appetite or eating
adiponectin
excessive appetite or eating
glycated hemoglobin
excessive appetite or eating
type 2 diabetes
excessive appetite or eating
metabolic syndrome
excessive appetite or eating
macrosomia
excessive appetite or eating
polyuria
excessive appetite or eating
hyperosmolar hyperglycemic syndrome
excessive appetite or eating
polyphagia
excessive appetite or eating
diabetic nephropathy
excessive appetite or eating
diabetic retinopathy
excessive appetite or eating
diabetic neuropathy
excessive appetite or eating
advanced glycation end products
excessive thirst
polydipsia
excessive thirst
type 1 diabetes
excessive thirst
prediabetes
excessive thirst
adiponectin
excessive thirst
glycated hemoglobin
excessive thirst
type 2 diabetes
excessive thirst
metabolic syndrome
excessive thirst
macrosomia
excessive thirst
polyuria
excessive thirst
hyperosmolar hyperglycemic syndrome
excessive thirst
polyphagia
excessive thirst
diabetic nephropathy
excessive thirst
diabetic retinopathy
excessive thirst
diabetic neuropathy
excessive thirst
advanced glycation end products
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
polydipsia
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
type 1 diabetes
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
prediabetes
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
adiponectin
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
glycated hemoglobin
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
type 2 diabetes
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
metabolic syndrome
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
macrosomia
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
polyuria
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
hyperosmolar hyperglycemic syndrome
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
polyphagia
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
diabetic nephropathy
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
diabetic retinopathy
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
diabetic neuropathy
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
advanced glycation end products
retinal damage associated with diabetes
polydipsia
retinal damage associated with diabetes
type 1 diabetes
retinal damage associated with diabetes
prediabetes
retinal damage associated with diabetes
adiponectin
retinal damage associated with diabetes
glycated hemoglobin
retinal damage associated with diabetes
type 2 diabetes
retinal damage associated with diabetes
metabolic syndrome
retinal damage associated with diabetes
macrosomia
retinal damage associated with diabetes
polyuria
retinal damage associated with diabetes
hyperosmolar hyperglycemic syndrome
retinal damage associated with diabetes
polyphagia
retinal damage associated with diabetes
diabetic nephropathy
retinal damage associated with diabetes
diabetic retinopathy
retinal damage associated with diabetes
diabetic neuropathy
retinal damage associated with diabetes
advanced glycation end products
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
polydipsia
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
type 1 diabetes
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
prediabetes
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
adiponectin
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
glycated hemoglobin
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
type 2 diabetes
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
metabolic syndrome
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
macrosomia
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
polyuria
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
hyperosmolar hyperglycemic syndrome
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
polyphagia
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
diabetic nephropathy
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
diabetic retinopathy
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
diabetic neuropathy
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
advanced glycation end products
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
polydipsia
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
type 1 diabetes
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
prediabetes
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
adiponectin
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
glycated hemoglobin
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
type 2 diabetes
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
metabolic syndrome
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
macrosomia
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
polyuria
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
hyperosmolar hyperglycemic syndrome
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
polyphagia
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
diabetic nephropathy
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
diabetic retinopathy
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
diabetic neuropathy
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
advanced glycation end products
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
polydipsia
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
type 1 diabetes
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
prediabetes
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
adiponectin
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
glycated hemoglobin
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
type 2 diabetes
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
metabolic syndrome
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
macrosomia
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
polyuria
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
hyperosmolar hyperglycemic syndrome
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
polyphagia
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
diabetic nephropathy
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
diabetic retinopathy
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
diabetic neuropathy
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
advanced glycation end products
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
polydipsia
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
type 1 diabetes
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
prediabetes
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
adiponectin
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
glycated hemoglobin
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
type 2 diabetes
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
metabolic syndrome
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
macrosomia
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
polyuria
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
hyperosmolar hyperglycemic syndrome
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
polyphagia
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
diabetic nephropathy
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
diabetic retinopathy
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
diabetic neuropathy
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
advanced glycation end products
usually results from insulin resistance coupled with insufficient insulin secretion
polydipsia
usually results from insulin resistance coupled with insufficient insulin secretion
type 1 diabetes
usually results from insulin resistance coupled with insufficient insulin secretion
prediabetes
usually results from insulin resistance coupled with insufficient insulin secretion
adiponectin
usually results from insulin resistance coupled with insufficient insulin secretion
glycated hemoglobin
usually results from insulin resistance coupled with insufficient insulin secretion
type 2 diabetes
usually results from insulin resistance coupled with insufficient insulin secretion
metabolic syndrome
usually results from insulin resistance coupled with insufficient insulin secretion
macrosomia
usually results from insulin resistance coupled with insufficient insulin secretion
polyuria
usually results from insulin resistance coupled with insufficient insulin secretion
hyperosmolar hyperglycemic syndrome
usually results from insulin resistance coupled with insufficient insulin secretion
polyphagia
usually results from insulin resistance coupled with insufficient insulin secretion
diabetic nephropathy
usually results from insulin resistance coupled with insufficient insulin secretion
diabetic retinopathy
usually results from insulin resistance coupled with insufficient insulin secretion
diabetic neuropathy
usually results from insulin resistance coupled with insufficient insulin secretion
advanced glycation end products
hormone produced by adipose cells that promotes insulin sensitivity
polydipsia
hormone produced by adipose cells that promotes insulin sensitivity
type 1 diabetes
hormone produced by adipose cells that promotes insulin sensitivity
prediabetes
hormone produced by adipose cells that promotes insulin sensitivity
adiponectin
hormone produced by adipose cells that promotes insulin sensitivity
glycated hemoglobin
hormone produced by adipose cells that promotes insulin sensitivity
type 2 diabetes
hormone produced by adipose cells that promotes insulin sensitivity
metabolic syndrome
hormone produced by adipose cells that promotes insulin sensitivity
macrosomia
hormone produced by adipose cells that promotes insulin sensitivity
polyuria
hormone produced by adipose cells that promotes insulin sensitivity
hyperosmolar hyperglycemic syndrome
hormone produced by adipose cells that promotes insulin sensitivity
polyphagia
hormone produced by adipose cells that promotes insulin sensitivity
diabetic nephropathy
hormone produced by adipose cells that promotes insulin sensitivity
diabetic retinopathy
hormone produced by adipose cells that promotes insulin sensitivity
diabetic neuropathy
hormone produced by adipose cells that promotes insulin sensitivity
advanced glycation end products
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
polydipsia
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
type 1 diabetes
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
prediabetes
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
adiponectin
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
glycated hemoglobin
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
type 2 diabetes
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
metabolic syndrome
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
macrosomia
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
polyuria
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
hyperosmolar hyperglycemic syndrome
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
polyphagia
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
diabetic nephropathy
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
diabetic retinopathy
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
diabetic neuropathy
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
advanced glycation end products
kidney damage associated with diabetes
polydipsia
kidney damage associated with diabetes
type 1 diabetes
kidney damage associated with diabetes
prediabetes
kidney damage associated with diabetes
adiponectin
kidney damage associated with diabetes
glycated hemoglobin
kidney damage associated with diabetes
type 2 diabetes
kidney damage associated with diabetes
metabolic syndrome
kidney damage associated with diabetes
macrosomia
kidney damage associated with diabetes
polyuria
kidney damage associated with diabetes
hyperosmolar hyperglycemic syndrome
kidney damage associated with diabetes
polyphagia
kidney damage associated with diabetes
diabetic nephropathy
kidney damage associated with diabetes
diabetic retinopathy
kidney damage associated with diabetes
diabetic neuropathy
kidney damage associated with diabetes
advanced glycation end products
nerve degeneration associated with diabetes
polydipsia
nerve degeneration associated with diabetes
type 1 diabetes
nerve degeneration associated with diabetes
prediabetes
nerve degeneration associated with diabetes
adiponectin
nerve degeneration associated with diabetes
glycated hemoglobin
nerve degeneration associated with diabetes
type 2 diabetes
nerve degeneration associated with diabetes
metabolic syndrome
nerve degeneration associated with diabetes
macrosomia
nerve degeneration associated with diabetes
polyuria
nerve degeneration associated with diabetes
hyperosmolar hyperglycemic syndrome
nerve degeneration associated with diabetes
polyphagia
nerve degeneration associated with diabetes
diabetic nephropathy
nerve degeneration associated with diabetes
diabetic retinopathy
nerve degeneration associated with diabetes
diabetic neuropathy
nerve degeneration associated with diabetes
advanced glycation end products
سؤال
Illustrate the acute complications of uncontrolled diabetes.
سؤال
Discuss how the intakes of protein, carbohydrate, fat, sodium, and alcohol are modified for a patient with diabetes.
سؤال
Discuss sick-day management for people with diabetes.
سؤال
Compare and contrast conventional and intensive therapies for type 1 diabetic patients, including the advantages and disadvantages of each.
سؤال
Discuss how diabetes develops as a consequence of pregnancy and the potential consequences for both mother and fetus.
سؤال
Describe the criteria that are currently used to diagnose diabetes.
سؤال
Describe the variations in insulin preparations available for the treatment of diabetes.
سؤال
Explain the relationship of body weight and type 2 diabetes.
سؤال
61-65. Short Case Study Questions  
Ruth Ann Glebowski is a 40-year-old woman. She has a medical history of gestational diabetes, high cholesterol, hypothyroidism, and depression. She is 5 ft 4 in. tall and weighs 190 lb. Recently she has made an appointment with her physician since she has been having excessive hunger and frequent urination.
In addition to anti-diabetic medications, what can Ruth Ann do to help control her blood glucose?

A) lose weight
B) try a high-fat diet
C) take calcium supplements
D) start glucagon injections
سؤال
61-65. Short Case Study Questions  
Ruth Ann Glebowski is a 40-year-old woman. She has a medical history of gestational diabetes, high cholesterol, hypothyroidism, and depression. She is 5 ft 4 in. tall and weighs 190 lb. Recently she has made an appointment with her physician since she has been having excessive hunger and frequent urination.
Ruth Ann meets with a registered dietitian and is educated on carbohydrate counting for meal planning. Which of the following snacks would be appropriate if she is allowed 30 grams of carbohydrates?

A) A small apple and 4 sandwich cream-cookies
B) A small granola bar and a medium peach
C) 1 English muffin with a tablespoon of butter and 3 chocolate kisses
D) 2 slices of bread and 2 tablespoons of grape jelly
سؤال
Describe the different methods of insulin delivery available to people with diabetes.
سؤال
Describe the different types of neuropathy that may develop in patients with diabetes.
سؤال
61-65. Short Case Study Questions  
Ruth Ann Glebowski is a 40-year-old woman. She has a medical history of gestational diabetes, high cholesterol, hypothyroidism, and depression. She is 5 ft 4 in. tall and weighs 190 lb. Recently she has made an appointment with her physician since she has been having excessive hunger and frequent urination.
Fifteen years pass and Ruth Ann is experiencing some visual impairments. Her eye doctor explains that it could be diabetic ______________

A) nephropathy
B) hypoglycemia
C) retinopathy
D) gastroparesis
سؤال
61-65. Short Case Study Questions  
Ruth Ann Glebowski is a 40-year-old woman. She has a medical history of gestational diabetes, high cholesterol, hypothyroidism, and depression. She is 5 ft 4 in. tall and weighs 190 lb. Recently she has made an appointment with her physician since she has been having excessive hunger and frequent urination.
Ruth Ann is diagnosed with type 2 diabetes. Which factors puts her at increased risk for diabetes?

A) Weight and gestational diabetes
B) Depression and middle age
C) high cholesterol and hypothyroidism
D) Hypothyroidism
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Deck 26: Diabetes Mellitus
1
Diabetic ketoacidosis is characterized by severe ketosis, acidosis, and ____.

A) renal failure
B) hallucinations
C) hyperglycemia
D) myocardial ischemia
C
2
What best describes polydipsia?

A) excessive thirst
B) excessive urination
C) increased glucose in the urine
D) excessive hunger
A
3
Normal fasting plasma glucose levels are approximately ____ mg/dL.

A) 60 to 74
B) 75 to 100
C) 110 to 125
D) 126 to 140
B
4
Which of the following is NOT a feature of type 2 diabetes?

A) insulin resistance
B) autoimmune disease
C) obesity is a causative factor
D) some cases require insulin therapy
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5
Which condition is a microvascular complication of diabetes?

A) peripheral vascular disease
B) diabetic retinopathy
C) intermittent claudication
D) hypertriglyceridemia
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6
People with type 1 diabetes need exogenous insulin because they ____.

A) have become insulin-resistant
B) have developed hyperinsulinemia
C) no longer synthesize insulin
D) digest insulin with gastrointestinal enzymes
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7
The renal threshold is described as ____.

A) a decrease in erythropoietin production because of insulin imbalance
B) nutrient depletion in the renal tubules caused from deficient insulin
C) hemoglobin's exposure to glucose within the kidneys
D) the concentration at which the kidneys begin to pass glucose into the urine
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8
The oral glucose tolerance test uses a glucose load of ____ grams.

A) 25
B) 50
C) 75
D) 100
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9
Glycosuria usually occurs when plasma glucose concentration exceeds ____ mg/dL.

A) 140
B) 160
C) 180
D) 200
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10
A fasting blood glucose level above ____ mg/dL is classified as diabetes.

A) 90
B) 100
C) 116
D) 126
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11
Which ethnic population has the lowest relative risk for developing type 2 diabetes?

A) Pacific Islanders
B) Asian Indians
C) European Americans
D) Native Americans
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12
Diabetic ketoacidosis can develop when blood glucose is greater than ____ mg/dL.

A) 250
B) 450
C) 650
D) 700
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13
About ____ percent of persons with diabetes are unaware that they have it.

A) 7
B) 17
C) 24
D) 37
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14
The pancreatic hormone that promotes gluconeogenesis in the liver is ____.

A) glycogen
B) estrogen
C) glucagon
D) leptin
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15
Type 1 diabetes accounts for ____ percent of diabetes cases in the United States.

A) 5 to 10
B) 15 to 25
C) 50 to 75
D) 90 to 95
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16
Using the "intensive therapy" approach, blood glucose is monitored at least ____ time(s) a day for people with type 1 diabetes.

A) one
B) two
C) three
D) four
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17
What is the standard treatment for hyperosmolar hyperglycemic syndrome?

A) intravenous fluid and electrolyte replacement and insulin therapy
B) intravenous administration of ketones
C) surgical resection of the pancreas
D) a low-carbohydrate, low-fat diet
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18
Which is NOT a sign or symptom of diabetic neuropathy?

A) Numbness or tingling in extremities
B) gastroparesis
C) bladder dysfunction
D) diarrhea
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19
A blood glucose level between 100 and 125 mg/dL after an 8-hour fast would be classified as ____.

A) impaired fasting glucose
B) hypoglycemia
C) impaired glucose tolerance
D) normal fasting glucose
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20
Symptoms of hypoglycemia include ____.

A) sweating and heart palpitations
B) increased thirst and polyuria
C) acetone breath
D) warm, flushed skin
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21
Mrs. Barclay has type 2 diabetes and you are preparing her for discharge. She tells you that she loves red beans and rice and knows that she must eliminate them from her diet because they will elevate her blood glucose level. You should explain to her that ____.

A) on a carbohydrate-counting meal plan, these foods in recommended portion sizes can fit within her carbohydrate allowance at meals
B) red beans are eliminated because they are high in complex carbohydrates
C) red beans are high in water-soluble fiber and should be avoided
D) peas are a better choice than red beans
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22
As per the carbohydrate-counting meal plan, how many grams of carbohydrates is in a snack that consists of 2 cups of ice cream and a small apple?

A) 60 grams
B) 75 grams
C) 45 grams
D) 15 grams
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23
The newborn of a mother with diabetes is at greater risk of which metabolic condition?

A) hypocalcemia
B) sepsis
C) transient tachypnea
D) congenital heart disease
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24
Short-acting insulin begins to act ____ after it is injected.

A) 15 to 20 minutes
B) 30 to 60 minutes
C) 1 to 3 hours
D) 2 to 4 hours
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25
Approximately ____ percent of nondiabetic women in the United States develop gestational diabetes.

A) 4 to 14
B) 14 to 24
C) 24 to 34
D) 34 to 44
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26
Hemoglobin A 1c can best be described as a(n) ____.

A) by-product of fat metabolism
B) reflection of glycemic control over the preceding 2 to 3 months
C) end-product of protein metabolism formed in the liver
D) summary of hemoglobin rates for type 1 diabetes
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27
Women with gestational diabetes are at greater risk of which condition later in life?

A) obesity
B) type 2 diabetes
C) metabolic syndrome
D) chronic kidney disease
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28
Insulin is most often administered by ____.

A) subcutaneous injection
B) oral administration
C) intramuscular injection
D) intravenous administration
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29
Which antidiabetic drug is classified as a biguanide?

A) acarbose
B) pramlintide
C) metformin
D) nateglinide
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30
The goal of diabetes treatment is an HbA 1c value under ____ percent.

A) 5
B) 7
C) 9
D) 10
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31
What is an advantage seen with intensive therapy for patients with type 1 diabetes?

A) delayed progression of retinopathy, nephropathy, and neuropathy
B) less weight gain
C) greater stability noted in fructosamine results
D) fewer incidents of severe hypoglycemia
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32
Which recommendation is not advised for pregnant women with diabetes?

A) insulin therapy
B) 10% weight loss
C) physical activity
D) low-carbohydrate breakfast
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33
What maternal complication has been associated with uncontrolled diabetes during pregnancy?

A) preeclampsia
B) obesity
C) hyperemesis gravidarum
D) miscarriage
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34
Hyperglycemia that results from the release of counterregulatory hormones following nighttime hypoglycemia is known as ____.

A) fasting hyperglycemia
B) rebound hyperglycemia
C) dawn phenomenon
D) nocturnal hyperglycemia
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35
What test may be used to determine glycemic control over the preceding 2- to 3-week period?

A) fructosamine
B) ketone
C) glucose tolerance
D) insulin antibody
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36
Mr. Lilly, a 42-year-old male, tells you that he used to take "pills" for his diabetes but is now taking insulin. What is the most likely reason that he no longer takes oral antidiabetic agents?

A) Oral agents are effective only in type 1 diabetes.
B) His body now stimulates enough insulin to meet his needs.
C) Oral agents suppress insulin release.
D) The oral agents were not effectively controlling his blood glucose levels.
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37
Mr. Jacobs has high blood lipids. The physician would instruct him to limit dietary intake of which substance as an added sweetener but not from whole foods (i.e., fruits and vegetables)?

A) fructose
B) sucrose
C) glucose
D) sucralose
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38
Which intervention is most appropriate as part of sick-day management of diabetes?

A) Discontinue all antidiabetic medications and insulin.
B) Measure blood glucose and urine ketones once a day.
C) Discontinue antidiabetic drugs and only use insulin.
D) Consume the usual diet if possible.
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39
As per the carbohydrate-counting meal plan, how many grams of carbohydrates is in 2 cups of brown rice and a cup of red beans?

A) 100 grams
B) 120 grams
C) 150 grams
D) 175 grams
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40
What is a metabolic effect that may occur with taking thiazolidinediones for glycemic control?

A) fluid retention
B) weight loss
C) allergic skin reactions
D) vitamin B 12 deficiency
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41
Which patient would have the most risk factors for gestational diabetes?

A) Rita, who has a prepregnancy body mass index (BMI) of 24
B) Zhu, who just moved to the United States from China
C) Amy, who once had an 8-pound baby
D) Sarah, whose father has diabetes
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42
An estimated ____ percent of adults in the United States meet the criteria for metabolic syndrome.

A) 6
B) 12
C) 23
D) 34
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43
Which antidiabetic drug is sometimes prescribed for type 2 diabetic women during pregnancy?

A) glimepiride
B) nateglinide
C) repaglinide
D) glyburide
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44
56-60. Short Case-Study Questions  
Michael Fern is a 21-year-old college student majoring in electrical engineering. His medical history is unremarkable. He comes to his family doctor for the second time this month because of symptoms of lethargy, fatigue, weight loss, and frequent thirst and urination.
After 6 months, Michael's endocrinologist orders an insulin pump. Which best describes the use of an insulin pump with type 1 diabetes?

A) Hyperglycemia is not a concern with the insulin pump.
B) Hypoglycemia can occur with insulin treatment.
C) Michael does not need to follow a modified diet.
D) Michael no longer needs to worry about exercising.
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45
Diagnostic criteria for metabolic syndrome include a fasting plasma glucose of ____ mg/dL or higher.

A) 80
B) 90
C) 100
D) 110
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46
Diagnostic criteria for metabolic syndrome include blood pressure of ____ mm Hg or higher.

A) 110/80
B) 130/85
C) 150/90
D) 170/95
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47
56-60. Short Case-Study Questions  
Michael Fern is a 21-year-old college student majoring in electrical engineering. His medical history is unremarkable. He comes to his family doctor for the second time this month because of symptoms of lethargy, fatigue, weight loss, and frequent thirst and urination.
The registered dietitian visits with Michael to instruct him on his diabetic diet. What is important for Michael to know about consuming alcohol?

A) He should not consume any alcohol.
B) He should consume food when he ingests alcoholic beverages to avoid hypoglycemia.
C) He should limit his food intake prior to consuming alcohol in order to avoid hyperglycemia.
D) Excessive alcohol intake will likely cause severe hypoglycemia.
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48
56-60. Short Case-Study Questions  
Michael Fern is a 21-year-old college student majoring in electrical engineering. His medical history is unremarkable. He comes to his family doctor for the second time this month because of symptoms of lethargy, fatigue, weight loss, and frequent thirst and urination.
Michael is rushed to the hospital after a simple blood test determines a blood glucose of 600 mg/dL. He is most likely suffering from ____.

A) hypoglycemia
B) hyperglycemia
C) dehydration
D) infection
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49
Diagnostic criteria for metabolic syndrome include waist circumference of greater than ____ inches in women.

A) 33
B) 35
C) 38
D) 40
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50
Inflammation of endothelial tissue, obesity, and insulin resistance can all contribute to blood clots by promoting the increased production of procoagulant proteins such as ____.

A) hirudin
B) fibrinogen
C) dicoumarol
D) andexanet alfa
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51
Macrosomia develops as a result of ____.

A) insulin's stimulatory effect on fat synthesis
B) excessive insulin production by the fetal pancreas
C) poor maternal kidney function due to excess glucose
D) the enzymatic glycation of serum proteins
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52
56-60. Short Case-Study Questions  
Michael Fern is a 21-year-old college student majoring in electrical engineering. His medical history is unremarkable. He comes to his family doctor for the second time this month because of symptoms of lethargy, fatigue, weight loss, and frequent thirst and urination.
Michael's ketoacidosis leads to a diagnosis of type 1 diabetes. Which medication will Michael need to take to treat his diabetes?

A) sulfonylureas
B) metformin
C) insulin injections
D) alpha-glucosidase inhibitors
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53
56-60. Short Case-Study Questions  
Michael Fern is a 21-year-old college student majoring in electrical engineering. His medical history is unremarkable. He comes to his family doctor for the second time this month because of symptoms of lethargy, fatigue, weight loss, and frequent thirst and urination.
Michael has been diagnosed with diabetic ketoacidosis. In addition to severely elevated blood glucose levels, what other symptom is associated with this condition?

A) fruity odor on the breath
B) fluid retention
C) high blood pressure
D) vision loss
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54
Regina is at her first prenatal visit after recently discovering that she is pregnant. She is a low-risk patient with no history of diabetes. At what point will her physician test Regina for gestational diabetes?

A) 12 to 15 weeks
B) 16 to 20 weeks
C) 24 to 28 weeks
D) 29 to 32 weeks
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55
Diagnostic criteria for metabolic syndrome include high-density lipoprotein (HDL) levels of less than ____ mg/dL in men.

A) 30
B) 35
C) 40
D) 50
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56
Diagnostic criteria for metabolic syndrome include very-low-density lipoprotein (VLDL) levels of ____ mg/dL or higher.

A) 100
B) 125
C) 150
D) 175
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57
Emily has gestational diabetes and a BMI of 33. What nutrition recommendation should be made for Emily for weight control during her pregnancy?

A) Strive to lose 11 to 14 lb by the end of her pregnancy.
B) Increase carbohydrate intake to 55 to 60% of total energy.
C) Reduce kcalorie intake by 30% to slow further weight gain.
D) Continue with a normal diet and strive to lose weight after pregnancy.
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58
How does obesity lead to the development of hypertriglyceridemia?

A) Insulin causes adipose cells to store more triglycerides.
B) HDL competes with VLDL for cholesterol binding sites.
C) The liver produces more VLDL in response to fatty acids released by insulin-resistant adipose cells.
D) Fatty acids in the bloodstream resist excretion by HDL.
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59
The hyperinsulinemia that typically accompanies obesity promotes sodium reabsorption in the kidneys, resulting in fluid retention and ____.

A) increased abdominal obesity
B) hypertension
C) atherosclerosis
D) hypertriglyceridemia
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60
What dietary recommendation is advised for individuals with hypertriglyceridemia?

A) Reduce intake of added sugars and refined grain products.
B) Decrease sodium intake and improve intake of dairy products.
C) E liminate polyunsaturated fatty acids in the diet.
D) Reduce kcalorie intake by 45% daily.
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61
61-65. Short Case Study Questions  
Ruth Ann Glebowski is a 40-year-old woman. She has a medical history of gestational diabetes, high cholesterol, hypothyroidism, and depression. She is 5 ft 4 in. tall and weighs 190 lb. Recently she has made an appointment with her physician since she has been having excessive hunger and frequent urination.
Ruth Ann's doctor can best evaluate long-term diabetic compliance by checking her ____.

A) fasting blood glucose
B) urine ketones
C) blood pressure
D) glycated hemoglobin
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62
Match between columns
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
polydipsia
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
type 1 diabetes
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
prediabetes
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
adiponectin
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
glycated hemoglobin
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
type 2 diabetes
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
metabolic syndrome
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
macrosomia
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
polyuria
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
hyperosmolar hyperglycemic syndrome
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
polyphagia
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
diabetic nephropathy
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
diabetic retinopathy
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
diabetic neuropathy
blood glucose levels are higher than normal but not high enough to be diagnosed as diabetes; considered a major risk factor for future cardiovascular disease
advanced glycation end products
excessive urine production
polydipsia
excessive urine production
type 1 diabetes
excessive urine production
prediabetes
excessive urine production
adiponectin
excessive urine production
glycated hemoglobin
excessive urine production
type 2 diabetes
excessive urine production
metabolic syndrome
excessive urine production
macrosomia
excessive urine production
polyuria
excessive urine production
hyperosmolar hyperglycemic syndrome
excessive urine production
polyphagia
excessive urine production
diabetic nephropathy
excessive urine production
diabetic retinopathy
excessive urine production
diabetic neuropathy
excessive urine production
advanced glycation end products
excessive appetite or eating
polydipsia
excessive appetite or eating
type 1 diabetes
excessive appetite or eating
prediabetes
excessive appetite or eating
adiponectin
excessive appetite or eating
glycated hemoglobin
excessive appetite or eating
type 2 diabetes
excessive appetite or eating
metabolic syndrome
excessive appetite or eating
macrosomia
excessive appetite or eating
polyuria
excessive appetite or eating
hyperosmolar hyperglycemic syndrome
excessive appetite or eating
polyphagia
excessive appetite or eating
diabetic nephropathy
excessive appetite or eating
diabetic retinopathy
excessive appetite or eating
diabetic neuropathy
excessive appetite or eating
advanced glycation end products
excessive thirst
polydipsia
excessive thirst
type 1 diabetes
excessive thirst
prediabetes
excessive thirst
adiponectin
excessive thirst
glycated hemoglobin
excessive thirst
type 2 diabetes
excessive thirst
metabolic syndrome
excessive thirst
macrosomia
excessive thirst
polyuria
excessive thirst
hyperosmolar hyperglycemic syndrome
excessive thirst
polyphagia
excessive thirst
diabetic nephropathy
excessive thirst
diabetic retinopathy
excessive thirst
diabetic neuropathy
excessive thirst
advanced glycation end products
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
polydipsia
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
type 1 diabetes
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
prediabetes
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
adiponectin
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
glycated hemoglobin
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
type 2 diabetes
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
metabolic syndrome
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
macrosomia
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
polyuria
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
hyperosmolar hyperglycemic syndrome
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
polyphagia
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
diabetic nephropathy
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
diabetic retinopathy
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
diabetic neuropathy
usually results from autoimmune destruction of pancreatic beta-cells; little or no insulin is produced in the pancreas
advanced glycation end products
retinal damage associated with diabetes
polydipsia
retinal damage associated with diabetes
type 1 diabetes
retinal damage associated with diabetes
prediabetes
retinal damage associated with diabetes
adiponectin
retinal damage associated with diabetes
glycated hemoglobin
retinal damage associated with diabetes
type 2 diabetes
retinal damage associated with diabetes
metabolic syndrome
retinal damage associated with diabetes
macrosomia
retinal damage associated with diabetes
polyuria
retinal damage associated with diabetes
hyperosmolar hyperglycemic syndrome
retinal damage associated with diabetes
polyphagia
retinal damage associated with diabetes
diabetic nephropathy
retinal damage associated with diabetes
diabetic retinopathy
retinal damage associated with diabetes
diabetic neuropathy
retinal damage associated with diabetes
advanced glycation end products
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
polydipsia
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
type 1 diabetes
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
prediabetes
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
adiponectin
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
glycated hemoglobin
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
type 2 diabetes
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
metabolic syndrome
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
macrosomia
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
polyuria
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
hyperosmolar hyperglycemic syndrome
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
polyphagia
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
diabetic nephropathy
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
diabetic retinopathy
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
diabetic neuropathy
compounds formed when glucose combines with proteins; can damage tissues and lead to diabetic complications
advanced glycation end products
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
polydipsia
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
type 1 diabetes
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
prediabetes
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
adiponectin
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
glycated hemoglobin
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
type 2 diabetes
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
metabolic syndrome
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
macrosomia
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
polyuria
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
hyperosmolar hyperglycemic syndrome
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
polyphagia
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
diabetic nephropathy
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
diabetic retinopathy
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
diabetic neuropathy
hemoglobin molecules with glucose attached; used to evaluate long-term glycemic control
advanced glycation end products
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
polydipsia
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
type 1 diabetes
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
prediabetes
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
adiponectin
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
glycated hemoglobin
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
type 2 diabetes
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
metabolic syndrome
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
macrosomia
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
polyuria
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
hyperosmolar hyperglycemic syndrome
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
polyphagia
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
diabetic nephropathy
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
diabetic retinopathy
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
diabetic neuropathy
newborns having an abnormally large body when born to mothers with uncontrolled diabetes
advanced glycation end products
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
polydipsia
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
type 1 diabetes
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
prediabetes
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
adiponectin
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
glycated hemoglobin
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
type 2 diabetes
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
metabolic syndrome
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
macrosomia
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
polyuria
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
hyperosmolar hyperglycemic syndrome
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
polyphagia
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
diabetic nephropathy
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
diabetic retinopathy
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
diabetic neuropathy
cluster of interrelated clinical symptoms, including central obesity, insulin resistance, high blood pressure, and abnormal blood lipids, which together increase cardiovascular disease risk two- to three-fold
advanced glycation end products
usually results from insulin resistance coupled with insufficient insulin secretion
polydipsia
usually results from insulin resistance coupled with insufficient insulin secretion
type 1 diabetes
usually results from insulin resistance coupled with insufficient insulin secretion
prediabetes
usually results from insulin resistance coupled with insufficient insulin secretion
adiponectin
usually results from insulin resistance coupled with insufficient insulin secretion
glycated hemoglobin
usually results from insulin resistance coupled with insufficient insulin secretion
type 2 diabetes
usually results from insulin resistance coupled with insufficient insulin secretion
metabolic syndrome
usually results from insulin resistance coupled with insufficient insulin secretion
macrosomia
usually results from insulin resistance coupled with insufficient insulin secretion
polyuria
usually results from insulin resistance coupled with insufficient insulin secretion
hyperosmolar hyperglycemic syndrome
usually results from insulin resistance coupled with insufficient insulin secretion
polyphagia
usually results from insulin resistance coupled with insufficient insulin secretion
diabetic nephropathy
usually results from insulin resistance coupled with insufficient insulin secretion
diabetic retinopathy
usually results from insulin resistance coupled with insufficient insulin secretion
diabetic neuropathy
usually results from insulin resistance coupled with insufficient insulin secretion
advanced glycation end products
hormone produced by adipose cells that promotes insulin sensitivity
polydipsia
hormone produced by adipose cells that promotes insulin sensitivity
type 1 diabetes
hormone produced by adipose cells that promotes insulin sensitivity
prediabetes
hormone produced by adipose cells that promotes insulin sensitivity
adiponectin
hormone produced by adipose cells that promotes insulin sensitivity
glycated hemoglobin
hormone produced by adipose cells that promotes insulin sensitivity
type 2 diabetes
hormone produced by adipose cells that promotes insulin sensitivity
metabolic syndrome
hormone produced by adipose cells that promotes insulin sensitivity
macrosomia
hormone produced by adipose cells that promotes insulin sensitivity
polyuria
hormone produced by adipose cells that promotes insulin sensitivity
hyperosmolar hyperglycemic syndrome
hormone produced by adipose cells that promotes insulin sensitivity
polyphagia
hormone produced by adipose cells that promotes insulin sensitivity
diabetic nephropathy
hormone produced by adipose cells that promotes insulin sensitivity
diabetic retinopathy
hormone produced by adipose cells that promotes insulin sensitivity
diabetic neuropathy
hormone produced by adipose cells that promotes insulin sensitivity
advanced glycation end products
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
polydipsia
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
type 1 diabetes
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
prediabetes
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
adiponectin
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
glycated hemoglobin
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
type 2 diabetes
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
metabolic syndrome
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
macrosomia
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
polyuria
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
hyperosmolar hyperglycemic syndrome
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
polyphagia
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
diabetic nephropathy
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
diabetic retinopathy
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
diabetic neuropathy
extreme hyperglycemia that is associated with hyperosmolar blood, dehydration, and altered mental status
advanced glycation end products
kidney damage associated with diabetes
polydipsia
kidney damage associated with diabetes
type 1 diabetes
kidney damage associated with diabetes
prediabetes
kidney damage associated with diabetes
adiponectin
kidney damage associated with diabetes
glycated hemoglobin
kidney damage associated with diabetes
type 2 diabetes
kidney damage associated with diabetes
metabolic syndrome
kidney damage associated with diabetes
macrosomia
kidney damage associated with diabetes
polyuria
kidney damage associated with diabetes
hyperosmolar hyperglycemic syndrome
kidney damage associated with diabetes
polyphagia
kidney damage associated with diabetes
diabetic nephropathy
kidney damage associated with diabetes
diabetic retinopathy
kidney damage associated with diabetes
diabetic neuropathy
kidney damage associated with diabetes
advanced glycation end products
nerve degeneration associated with diabetes
polydipsia
nerve degeneration associated with diabetes
type 1 diabetes
nerve degeneration associated with diabetes
prediabetes
nerve degeneration associated with diabetes
adiponectin
nerve degeneration associated with diabetes
glycated hemoglobin
nerve degeneration associated with diabetes
type 2 diabetes
nerve degeneration associated with diabetes
metabolic syndrome
nerve degeneration associated with diabetes
macrosomia
nerve degeneration associated with diabetes
polyuria
nerve degeneration associated with diabetes
hyperosmolar hyperglycemic syndrome
nerve degeneration associated with diabetes
polyphagia
nerve degeneration associated with diabetes
diabetic nephropathy
nerve degeneration associated with diabetes
diabetic retinopathy
nerve degeneration associated with diabetes
diabetic neuropathy
nerve degeneration associated with diabetes
advanced glycation end products
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63
Illustrate the acute complications of uncontrolled diabetes.
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64
Discuss how the intakes of protein, carbohydrate, fat, sodium, and alcohol are modified for a patient with diabetes.
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65
Discuss sick-day management for people with diabetes.
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66
Compare and contrast conventional and intensive therapies for type 1 diabetic patients, including the advantages and disadvantages of each.
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67
Discuss how diabetes develops as a consequence of pregnancy and the potential consequences for both mother and fetus.
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68
Describe the criteria that are currently used to diagnose diabetes.
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69
Describe the variations in insulin preparations available for the treatment of diabetes.
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70
Explain the relationship of body weight and type 2 diabetes.
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71
61-65. Short Case Study Questions  
Ruth Ann Glebowski is a 40-year-old woman. She has a medical history of gestational diabetes, high cholesterol, hypothyroidism, and depression. She is 5 ft 4 in. tall and weighs 190 lb. Recently she has made an appointment with her physician since she has been having excessive hunger and frequent urination.
In addition to anti-diabetic medications, what can Ruth Ann do to help control her blood glucose?

A) lose weight
B) try a high-fat diet
C) take calcium supplements
D) start glucagon injections
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72
61-65. Short Case Study Questions  
Ruth Ann Glebowski is a 40-year-old woman. She has a medical history of gestational diabetes, high cholesterol, hypothyroidism, and depression. She is 5 ft 4 in. tall and weighs 190 lb. Recently she has made an appointment with her physician since she has been having excessive hunger and frequent urination.
Ruth Ann meets with a registered dietitian and is educated on carbohydrate counting for meal planning. Which of the following snacks would be appropriate if she is allowed 30 grams of carbohydrates?

A) A small apple and 4 sandwich cream-cookies
B) A small granola bar and a medium peach
C) 1 English muffin with a tablespoon of butter and 3 chocolate kisses
D) 2 slices of bread and 2 tablespoons of grape jelly
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73
Describe the different methods of insulin delivery available to people with diabetes.
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74
Describe the different types of neuropathy that may develop in patients with diabetes.
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75
61-65. Short Case Study Questions  
Ruth Ann Glebowski is a 40-year-old woman. She has a medical history of gestational diabetes, high cholesterol, hypothyroidism, and depression. She is 5 ft 4 in. tall and weighs 190 lb. Recently she has made an appointment with her physician since she has been having excessive hunger and frequent urination.
Fifteen years pass and Ruth Ann is experiencing some visual impairments. Her eye doctor explains that it could be diabetic ______________

A) nephropathy
B) hypoglycemia
C) retinopathy
D) gastroparesis
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76
61-65. Short Case Study Questions  
Ruth Ann Glebowski is a 40-year-old woman. She has a medical history of gestational diabetes, high cholesterol, hypothyroidism, and depression. She is 5 ft 4 in. tall and weighs 190 lb. Recently she has made an appointment with her physician since she has been having excessive hunger and frequent urination.
Ruth Ann is diagnosed with type 2 diabetes. Which factors puts her at increased risk for diabetes?

A) Weight and gestational diabetes
B) Depression and middle age
C) high cholesterol and hypothyroidism
D) Hypothyroidism
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فتح الحزمة
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