A toxin that binds specifically to voltage- gated sodium channels in axons would be expected to
A) prevent the hyperpolarization phase of the action potential.
B) prevent the depolarization phase of the action potential.
C) increase the release of neurotransmitter molecules.
D) have most of its effects on the dendritic region of a neuron.
E) prevent graded potentials.

Question

Quizplus · Accepted Answer

Voltage-gated sodium channels are responsible for the depolarization phase of the action potential, so a toxin that binds specifically to these channels would be expected to prevent the depolarization phase of the action potential. This would lead to a disruption of neuronal communication and potentially result in paralysis or other neurological deficits.

A Toxin That Binds Specifically to Voltage- Gated Sodium Channels