Disner et al. (2011) proposed a neurobiological theory of depression based on a presumed role that the amygdala plays in relation to memory. Specifically, they have argued that the amygdala:
A) is larger in people who are depressed therefore threat-cues are more likely to be incorporated into stored memories.
B) becomes hyperactivated in depression, thereby causing connected areas like the hippocampus to become more activated and thus more likely to recall negative events.
C) is smaller in people who are depressed therefore reward-cues are less likely to be incorporated into stored memories.
D) becomes inhibited in depression, thereby causing connected areas like the hippocampus to become less activated and thus less likely to recall memories of any sort.
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