Endometrial hyperplasia is typically a result of prolonged exposure to:

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Endometrial hyperplasia is typically a result of prolonged exposure to estrogen, as it promotes the growth and proliferation of the endometrial cells. Lack of progesterone can also contribute to the development of endometrial hyperplasia, as progesterone triggers the shedding of the endometrial lining during menstruation, and its absence can lead to overgrowth of the cells. Vitamin A, Vitamin D, and thyroid hormones do not have a direct role in the development of endometrial hyperplasia.

Endometrial Hyperplasia Is Typically a Result of Prolonged Exposure To