The pathogenesis of polycystic ovarian syndrome is described as:
A) a decrease in leptin levels that reduces the hypothalamic pulsatility of gonadotropin-releasing hormone, which reduces the number of follicles that mature.
B) a disorder in the anterior pituitary that increases the follicle-stimulating hormone, which reduces the luteinizing hormone released.
C) excessive androgens that affect follicular decline by suppressing apoptosis, enabling follicles, which normally disintegrate to survive.
D) testosterone that stimulates androgen secretion by the ovarian stroma and reduces sex hormone-binding globulin indirectly.
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