How does onabotulinumtoxinA (BOTOX) therapeutically produce paresis in overactive muscles?
A) Acts as an antagonist by binding to the ACh receptor on the postsynaptic membrane.
B) Rapidly degrades ACh in the synaptic cleft.
C) Facilitates the reuptake and sequestration of ACh into the presynaptic cell.
D) Disrupts the protein structure of the muscle cell receptor,thus preventing ACh from binding.
E) Inhibits the release of ACh from the presynaptic terminal at the neuromuscular junction.
Correct Answer:
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Q8: Which of the following is associated with
Q9: Myasthenia gravis:
A)Is caused by the destruction of
Q10: An antagonist drug acts by:
A)Preventing the release
Q11: Which one of the following can serve
Q12: The binding of ACh at the neuromuscular
Q14: Which of the following is a type
Q15: Receptor tyrosine kinases:
A)Act through second messenger systems.
B)Are
Q16: N-methyl-D-aspartate (NMDA)receptors:
A)Are involved in long-term potentiation.
B)Bind glutamate.
C)Have
Q17: Second messengers may initiate the:
A)Opening of membrane
Q18: Substance P is an example of a(n):
A)Amino
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