The genetic disorder sickle-cell anemia develops because the hemoglobin molecule is deformed.Afflicted individuals often suffer muscle pain,even at low levels of exertion.A reasonable hypothesis for the symptoms is that the deformed hemoglobin
A) is unable to carry the amount of oxygen required for normal muscle activity.
B) cannot diffuse across the red blood cell membranes to the blood plasma,causing an oxygen shortage.
C) causes damage to muscle proteins as it diffuses into the muscle cell.
D) damages the alveolar surfaces and decreases the total surface area available for gas exchange.

Question

Quizplus · Accepted Answer

The deformed hemoglobin in sickle-cell anemia does not carry oxygen effectively, leading to a shortage of oxygen in the affected tissues, including muscle. This oxygen shortage can result in muscle pain and fatigue, especially during exertion.

The Genetic Disorder Sickle-Cell Anemia Develops Because the Hemoglobin Molecule