The alternative and classical pathway C3 convertases are destabilized as a means of downregulating complement function.One molecule that performs this function is:

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Membrane cofactor protein (MCP or CD46) is a glycoprotein that serves as a cofactor for the degradation of C3b by factor I. This accelerates the decay of C3 convertases, including the alternative and classical pathways, thereby downregulating complement function. MCP is expressed on all human nucleated cells and helps to protect host cells from complement-mediated damage.

The Alternative and Classical Pathway C3 Convertases Are Destabilized as a Means