In familial hypercholesterolemia, individuals who are homozygous for the mutation lack LDL cholesterol receptors in the liver, causing high levels of serum cholesterol. This is a defect in which type of gene pathway: a biosynthetic, signal transduction, or developmental pathway?

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The Answer of In familial hypercholesterolemia, individuals who are homozygous for the mutation lack LDL cholesterol receptors in the liver, causing high levels of serum cholesterol. This is a defect in which type of gene pathway: a biosynthetic, signal transduction, or developmental pathway?

In Familial Hypercholesterolemia, Individuals Who Are Homozygous for the Mutation