Salbutamol is an epinephrine agonist for the $\beta$-adrenergic receptor. What effects would you expect to see if you treat a hepatocyte with salbutamol?
A) inactivation of adenylylcyclase
B) activation of glycolysis
C) activation of glycogenolysis
D) glucose export from cells
E) both activation of glycogenolysis and glucose export from cells

Question

Quizplus · Accepted Answer

Salbutamol, being a β-adrenergic agonist, stimulates the β-adrenergic receptors leading to the activation of adenylyl cyclase, which in turn increases cAMP levels. This cascade activates protein kinase A (PKA), which then activates glycogenolysis (the breakdown of glycogen to glucose) and facilitates glucose export from cells to meet energy demands.

Salbutamol Is an Epinephrine Agonist for The β\betaβ -Adrenergic Receptor

Salbutamol Is an Epinephrine Agonist for The $\beta$ -Adrenergic Receptor